澳洲雪梨大学论文代写：遗传基因会影响乳腺癌吗?
TP53基因为人体提供方向，使其产生阻止肿瘤生长的蛋白质。TP53基因异常是Li-Fraumeni综合征的主要原因，Li-Fraumeni综合征是一种疾病，可导致个人在年轻时罹患软组织癌。患有这种综合症的人患乳腺癌和脑瘤、肉瘤和白血病等许多其他癌症的风险更高。BRIP1和BRCA基因遗传了MRE11A、PALB2、NBN、RAD51或RAD50的异常拷贝，导致个体发生范可尼anema疾病，抑制骨髓功能，导致白细胞、血小板和红细胞水平下降(rose, 2005)。人体内的范可尼贫血会增加罹患脑癌和肾癌等不同类型癌症的风险。遗传顾问命令测试异常CDH1、ATM、MRE11A、CHEK2、PLAB2、NBN、RAD50、PTEN、TP53和RAD51C基因，这些基因是包括BRAC2和BRAC1在内的较大面板基因的主要组成部分。异常基因的增加增加了患乳腺癌的风险。科学家们正在对乳腺癌的治疗进行投资。
澳洲雪梨大学论文代写：遗传基因会影响乳腺癌吗?
The TP53 genes provide directions to the body for producing proteins that prevent the growth of the tumour. The abnormal TP53 genes are the main cause of Li-Fraumeni syndrome which is a disorder that leads to the development of soft tissue cancers in an individual at a young age. An individual with this syndrome has the higher risk of breast cancer and many other cancers such as brain tumours, sarcomas and leukemia. Inheriting the abnormal copies of the MRE11A, PALB2, NBN, RAD51 or RAD50, BRIP1 and BRCA genes cause the Fanconi anema disease in an individual which suppresses the functions of bone marrow and leads to decrease levels of the white blood cells, platelets and red blood cells (Roses, 2005). The Fanconi anemia in the body of an individual increases the risk of different types of cancer such as brain cancer and kidney cancer. A genetic counsellor orders to test the abnormal CDH1, ATM, MRE11A, CHEK2, PLAB2, NBN, RAD50, PTEN, TP53 and RAD51C genes which are the major part of the larger panel genes that include BRAC2 and BRAC1. The increase in abnormal genes increases the risk of breast cancer. The scientists are investing on the treatments of the breast cancer.
