β-肾上腺素能受体有助于结合平滑肌组织上的受体。 β-肾上腺素能受体使用通常由交感神经雄激素刺激释放。同样的另一个影响是，它可能导致在节点组织和传导系统中的危害。这也导致可能在血液中循环的去甲肾上腺素和肾上腺素的结合。受体也可能表现出可能被证明与α受体不同的一组反应。这些是由于其在体内对异丙肾上腺素的反应而可能表征的那些。根据情况可以考虑对肾上腺素能刺激的过程使用适当的心理反应。可能会有同样的刺激。可以发现肾上腺素和去甲肾上腺素的亲和力与α1和α2的亲和力相同。尽管在其他一些情况下，我发现其中一种药物的亲和力比受体的亲和力要高得多（Peuler等625）。这些价值亲和力的差异已被完全用于将受体分为两种不同的类型。已经发现，肾上腺素通常具有较高的与α1受体中β2沉降有关的亲和力值。因此，肾上腺素对平滑肌的影响可能是理想的，因为对α1和β2受体可能存在相对亲和力的依赖性较低。每当肾上腺素的浓度较低时，可导致β2 – 受体的选择性刺激。因此，当肾上腺素浓度增加时，血管收缩可能是主要的影响。
Beta-adrenoceptors helps in the process of binding the receptors which are on the smooth muscle tissues. Beta-adrenoceptor use is generally released by the sympathetic androgenic stimulation. Another effect of the same is that it can lead to the harms in the nodal tissue and the conducting systems. These also lead to the binding of both the norepinephrine and epinephrine which may be circulating in the blood. The receptors may also be exhibiting the set of responses which may prove to be the different from those of α receptors. These are the ones that may be characterized because of its response to isoproterenol in the body. It may be considered to use the proper psychological responses to the process of adrenergic stimulation as per the kind of situations. There may be stimulations of the same. It can be found that affinity for Epinephrine and norepinephrine is same as that of the affinity of alpha¬1 and alpha¬¬2. Though, in some other instances I have been found that the affinity of one of the drug had been much higher in comparison to that of the receptor (Peuler et al 625). These kinds of differences in the value affinity had been completely used for the sub classification of the receptors into the two different types. It has been found that epinephrine generally has a higher value of affinity related to the beta2 settle in some of the alpha1 receptors. Thus, it can be idealized that the impact of epinephrine on the smooth muscles is because of the lower dependence on the relative affinity which may be there for the alpha¬¬¬¬1 and beta2 receptors. Whenever the concentration of epinephrine is lower, it can lead to the selective stimulation of the beta2 ¬receptors. Thus, when the concentration of epinephrine increases, there may be a predominant impact of vasoconstriction.